Rabeprazole Sodium or (Rabeprazole na) a proton pump inhibitor (PPI) that suppresses gastric acid secretion by inhibiting the H⁺/K⁺-ATPase enzyme system at the secretory surface of gastric parietal cells action that leads to a reduction in gastric acidity, providing relief from acid-related disorders. Rabeprazole treats gastroesophageal reflux disease (GERD), peptic ulcers, and Zollinger-Ellison syndrome. The compound exhibits hygroscopic properties to be specific. Rabeprazole sodium, also known as Rabeprazole sodium salt, is preferred by manufacturers in the pharmaceutical industry for the production of oral dosage forms, such as enteric-coated tablets and capsules, designed to treat acid-related gastrointestinal disorders. Rabeprazole Sodium is a Proton Pump Inhibitor (PPI); Acid-Suppressing Agent; Delayed-Release API for GI Disorders.
Rabeprazole Sodium or (Rabeprazole na) a proton pump inhibitor (PPI) that suppresses gastric acid secretion by inhibiting the H⁺/K⁺-ATPase enzyme system at the secretory surface of gastric parietal cells action that leads to a reduction in gastric acidity, providing relief from acid-related disorders. Rabeprazole treats gastroesophageal reflux disease (GERD), peptic ulcers, and Zollinger-Ellison syndrome. The compound exhibits hygroscopic properties to be specific. Rabeprazole sodium, also known as Rabeprazole sodium salt, is preferred by manufacturers in the pharmaceutical industry for the production of oral dosage forms, such as enteric-coated tablets and capsules, designed to treat acid-related gastrointestinal disorders. Rabeprazole Sodium is a Proton Pump Inhibitor (PPI); Acid-Suppressing Agent; Delayed-Release API for GI Disorders.
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Pharmaceutical
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Pharmaceutical Actives & Precursors
Active Pharmaceutical Ingredients (APIs)
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Chemical Properties & Specifications
Used to alleviate symptoms and promote healing in patients with GERD by reducing gastric acid secretion.
Aids in the healing of duodenal and gastric ulcers by inhibiting acid production.
Employed as part of combination therapy to eliminate H. pylori, reducing the risk of ulcer recurrence.
Manages pathological hypersecretory conditions by suppressing excessive gastric acid production.
Commonly formulated in enteric-coated tablets to ensure stability and targeted release in the intestine.
Irreversibly binds to and inhibits the H⁺/K⁺-ATPase enzyme system, leading to prolonged suppression of gastric acid secretion.
Approximately 52% following oral administration.
Approximately 96.3%.
Extensively metabolized in the liver via CYP2C19 and CYP3A4 isoenzymes.
Approximately 1 hour.
Primarily excreted in urine as metabolites.
Pharmacopoeial Standards: Complies with USP, EP, and BP specifications.
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